Share your insights: The Collection from the International Conference for the Developmental Biology of the Sea Urchin and Marine Invertebrates in Discover Developmental Biology is now open for submissions

Share your insights: The Collection from the International Conference for the Developmental Biology of the Sea Urchin and Marine Invertebrates in Discover Developmental Biology is now open for submissions

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The DBSUMI XXIX Conference Collection: Selected Papers from the International Conference for the Developmental Biology of the Sea Urchin and Marine Invertebrates

Discover Developmental Biology is please to collaborate with the 2026 International Conference for the Developmental Biology of the Sea Urchin and Marine Invertebrates (DBSUMI XXIX, taking place from 7 to 11 October 2026 at the Marine Biological Laboratory) to launch a new Conference Collection, inviting all participants of DBSUMI XXIX to contribute their latest research.

What is this collection about?

For over a century, echinoderm embryos have served as foundational model organisms in developmental biology. Research in echinoderms has yielded critical insights into gene regulatory network architecture, cell lineage specification, and the dynamic morphogenetic processes, driving early embryonic development. Recent transgenic creation of several of these echinoderm species further provide unprecedented research tools for making exciting discoveries in developmental processes.

This Collection invites participants of the DBSUMI XXIX conference to submit research, review, methodology, brief report or perspective articles. Topics span from reproduction, cell fate specification, gene regulatory mechanisms, signaling pathways, regeneration, and organogenesis.

The submission deadline will be 31 July 2027.

Championing the conference collection

Dr. Mamiko Yajima is an Associate Professor (Research) in the Division of Biology and Medicine, Department of Molecular Biology, Cell Biology, and Biochemistry at Brown University, USA. Her research focuses on the mechanisms of asymmetric cell division and molecular segregation that contribute to asymmetric cell fate during early embryogenesis, using sea urchin embryos as a major model system. Dr. Yajima will serve as Lead Guest Editor for this collection.

Prof. Smadar Ben-Tabou de-Leon obtained her PhD in theoretical physics from the Hebrew University of Jerusalem and later transitioned to developmental and evolutionary biology during postdoctoral research at the Davidson lab at Caltech. She is an Associate Professor and Head of the Department of Marine Biology at the University of Haifa. Her research uses sea urchin embryos to study the biological regulation and evolution of development. Dr. Ben-Tabou de-Leon is one of the co-Guest Editors of this collection.

Dr. Gary Wessel is Professor of Biology at Brown University, experienced teacher and researcher with over 250 peer reviewed publications, former Editor-in-Chief and Editorial Board member to several journals in the area of reproduction and development. Dr. Wessel will co-lead this collection alongside the other Guest Editors.

Dr. Rossella Annunziata is an evolutionary developmental biologist. She earned her PhD at the Stazione Zoologica Anton Dohrn (SZN, Naples), where she investigated the evolution of gut patterning Gene Regulatory Networks in echinoderms. In 2019, she joined SZN as a researcher in the Department of Biology and Evolution of Marine Organisms. Dr. Annunziata is one of the co-Guest Editors of this collection.

How can I submit my paper?

Visit the Collection page to find out more about this collection and submit your article.

Follow the Topic

Developmental Biology and Stem Cells
Life Sciences > Biological Sciences > Developmental Biology and Stem Cells
Embryogenesis
Life Sciences > Biological Sciences > Developmental Biology and Stem Cells > Embryology > Embryogenesis
Cell Biology
Life Sciences > Biological Sciences > Cell Biology
Organogenesis
Life Sciences > Biological Sciences > Developmental Biology and Stem Cells > Organogenesis
Developmental Genetics
Life Sciences > Biological Sciences > Genetics and Genomics > Developmental Genetics
Evolutionary Developmental Biology
Life Sciences > Biological Sciences > Evolutionary Biology > Evolutionary Developmental Biology

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Molecular Control of Cardiac Development and Congenital Heart Disease

This proposed collection will highlight recent advances in understanding the molecular mechanisms that regulate heart development and how their disruption leads to congenital heart disease. We welcome original research, reviews, and perspectives addressing genetic, epigenetic, transcriptional, and signaling pathways that govern cardiac cell specification, morphogenesis, chamber formation, and functional maturation. Particular interest will be given to studies that connect developmental mechanisms with disease pathogenesis, including work using animal models, stem cell systems, human genetics, and multi-omics approaches. By bringing together studies across basic and translational developmental biology, this collection aims to provide new insights into the origins of congenital heart disease and to promote emerging strategies for diagnosis, modeling, and therapeutic intervention.

Keywords: cardiac development, congenital heart disease, transcriptional regulation, epigenetics, cardiogenesis, animal models

Publishing Model: Open Access

Deadline: Jan 29, 2027

Developmental Programming and Epigenetics: Mechanisms, Implications, and Interventions

Developmental programming refers to the process by which early-life environmental factors influence the long-term health outcomes of an individual, including susceptibility to diseases such as cardiovascular disease, diabetes, and cancer. Epigenetic modifications, which involve changes in gene expression without altering the underlying DNA sequence, have been increasingly recognized as key mediators of developmental programming. These modifications include DNA methylation, histone modifications, and non-coding RNAs, and can be influenced by a variety of factors such as stress, nutrition, toxins, and other environmental exposures during critical periods of development. Notably, epigenetic changes may be heritable, leading to transgenerational effects on health.

This Collection aims to explore the growing body of research examining the connection between developmental programming and epigenetics, with a focus on how exposures during critical period of development can impact gene expression and contribute to disease risk across the lifespan. We invite contributions that advance our understanding of the mechanisms underlying developmental programming and epigenetic regulation, and that offer insights into potential therapeutic approaches for mitigating adverse health outcomes linked to early-life exposures.

Potential topics for this Collection include, but are not limited to:

Mechanisms of developmental programming: How early-life environmental exposures (e.g., nutrition, toxins, stress) induce epigenetic changes and influence long-term health.

DNA methylation and histone modification in developmental programming: Insights into how these epigenetic marks regulate gene expression during critical periods of development.

Transgenerational epigenetic inheritance: Evidence and mechanisms by which epigenetic changes are passed from one generation to the next, affecting offspring health.

The role of non-coding RNAs in developmental programming and epigenetic regulation.

The impact of prenatal and early-life exposures on metabolic programming, neurodevelopment, and immune system function.

The influence of the maternal environment on epigenetic modifications and developmental outcomes.

The potential of epigenetic biomarkers in predicting disease risk and therapeutic interventions for early-life programming.

Epigenetic plasticity and interventions to mitigate the impact of harmful exposures (e.g., lifestyle changes, pharmacological approaches, and environmental modifications).

Keywords: developmental programming, epigenetic modifications, early-life exposures, transgenerational inheritance, disease risk

Publishing Model: Open Access

Deadline: Nov 04, 2026