Not All FoxP3-Positive Lymphocytes Are Equal: Spatial Localization Predicts Survival in Pancreatic Cancer

In pancreatic cancer, location matters. We explored whether the spatial distribution of FoxP3-positive lymphocytes within the tumor microenvironment provides prognostic information and influences survival outcomes in resected PDAC.
Not All FoxP3-Positive Lymphocytes Are Equal: Spatial Localization Predicts Survival in Pancreatic Cancer
Like

Share this post

Choose a social network to share with, or copy the URL to share elsewhere

This is a representation of how your post may appear on social media. The actual post will vary between social networks

Explore the Research

Springer US
Springer US Springer US

Spatially resolved FoxP3 positive lymphocyte infiltration within the tumor microenvironment predicts survival in resected pancreatic ductal adenocarcinoma - Discover Oncology

Objective In pancreatic ductal adenocarcinoma (PDAC), immune and stromal components of the tumor microenvironment critically influence disease progression and survival. Although FoxP3 expression has been linked to prognosis, the clinical relevance of its spatial distribution remains unclear. This study aimed to evaluate the prognostic significance of FoxP3-positive lymphocytes in intratumoral (IT), peritumoral (PT), and tumor-associated stromal (T) compartments. Materials and methods Ninety-eight patients with PDAC who underwent surgical resection between 2015 and 2021 were retrospectively analyzed. FoxP3 expression was assessed immunohistochemically in IT, PT, and T compartments and categorized as low or high based on H-scores. Associations with clinicopathological variables, overall survival (OS), and disease-free survival (DFS) were analyzed using Kaplan–Meier and Cox proportional hazards models. Subgroup analyses were performed according to recurrence status and receipt of adjuvant chemotherapy. Results High FoxP3 expression in IT, PT, and T compartments was significantly associated with shorter OS. Although FoxP3 expression showed significant associations with DFS in Kaplan–Meier analyses, it did not retain independent significance in multivariate models. Tumor-associated stromal FoxP3 expression correlated with increased stromal response and advanced nodal stage, whereas high PT-FoxP3 expression was associated with positive surgical margins. In patients receiving adjuvant chemotherapy, Kaplan–Meier analyses showed no clear survival separation; however, compartment-specific prognostic effects of FoxP3 were preserved in multivariate analyses. Conclusion FoxP3-positive lymphocyte infiltration represents a clinically relevant prognostic biomarker in PDAC, with its impact on survival strongly dependent on spatial localization within the tumor microenvironment. Compartment-based evaluation of FoxP3 may improve risk stratification and provide a biologically meaningful framework for future immunologically informed therapeutic strategies in PDAC.

Representative FoxP3 immunohistochemical staining in pancreatic ductal adenocarcinoma demonstrating high (A), moderate (B), and low (C) lymphocyte infiltration within the tumor microenvironment.
Figure 1. Representative FoxP3 immunohistochemical staining patterns demonstrating strong, moderate, and weak expression across intratumoral (IT), peritumoral (PT), and tumor-associated stromal (T) compartments.Caption

Why Study FoxP3?

FoxP3-positive lymphocytes are commonly associated with regulatory T-cell activity and immune suppression. Although several studies have examined FoxP3 in pancreatic cancer, most evaluated it as a single measurement without considering where these immune cells were located.

We hypothesized that the biological significance of FoxP3-positive lymphocytes might depend on their spatial distribution within the tumor microenvironment.

To investigate this question, we analyzed 98 surgically resected PDAC cases and separately assessed FoxP3 expression in three distinct compartments:

  • Intratumoral (IT)
  • Peritumoral (PT)
  • Tumor-associated stromal (T)

This compartment-based approach enabled a more detailed characterization of the immune landscape surrounding pancreatic cancer.

High FoxP3 Expression Predicts Poorer Overall Survival

One of the most striking findings of our study was the consistent association between elevated FoxP3 expression and reduced overall survival.

Kaplan–Meier overall survival curves demonstrating worse survival in pancreatic ductal adenocarcinoma patients with high FoxP3 expression compared with low FoxP3 expression across stromal, peritumoral, and intratumoral compartments.
Figure 6. Kaplan–Meier overall survival curves according to FoxP3 expression in intratumoral, peritumoral, and tumor-associated stromal compartments

Across all evaluated compartments, patients with high FoxP3 expression demonstrated significantly shorter overall survival than those with low expression.

These findings suggest that immunosuppressive immune populations contribute to aggressive tumor behavior regardless of their precise anatomical location.

High FoxP3 Expression Also Predicts Earlier Recurrence

Overall survival represents only one aspect of clinical outcome. We therefore evaluated disease-free survival to determine whether FoxP3-positive lymphocytes were also associated with tumor recurrence.

ROC curves demonstrating the diagnostic performance of intratumoral, peritumoral, and stromal FoxP3 expression in pancreatic ductal adenocarcinoma. Sensitivity is plotted against specificity, with a cut-off value of 100 used for classification.

Figure 8. Kaplan–Meier disease-free survival curves according to FoxP3 expression.

Patients exhibiting higher FoxP3 expression experienced earlier recurrence following surgical resection. This finding suggests that FoxP3-associated immune suppression may influence not only mortality but also mechanisms driving disease progression and relapse.

 

Prognostic Effects Persist in Patients Receiving Adjuvant Therapy

An important question was whether FoxP3 retained prognostic significance among patients receiving postoperative chemotherapy.

Prognostic Impact of Compartment-Specific FoxP3 Expression on Overall and Disease-Free Survival
Figure 12. Forest plot demonstrating compartment-specific prognostic effects of FoxP3 expression in patients receiving adjuvant chemotherapy.

Interestingly, compartment-specific FoxP3 expression remained associated with survival outcomes even within this clinically important subgroup. These findings support the robustness of spatial immune profiling and suggest that FoxP3-related biology may remain relevant despite systemic treatment.

 

Why Spatial Pathology Matters

Traditional pathology has always relied on spatial information. Pathologists routinely evaluate tumor invasion, lymphovascular invasion, surgical margins, and many other features based on anatomical relationships.

Our findings suggest that immune biomarkers should be approached similarly.

Rather than simply determining whether immune cells are present, future pathology workflows may increasingly focus on where these cells are located and how they interact with surrounding structures.

Advances in digital pathology, artificial intelligence, multiplex imaging, and spatial transcriptomics may further enhance our ability to characterize these complex relationships.

 

Key Findings

🔬 98 resected PDAC patients were evaluated.

📍 FoxP3-positive lymphocytes were assessed separately in intratumoral, peritumoral, and stromal compartments.

📈 High FoxP3 expression predicted significantly shorter overall survival.

📉 High FoxP3 expression predicted shorter disease-free survival.

🧬 Spatial localization provided clinically meaningful prognostic information.

🚀 Spatial pathology approaches may improve future biomarker development and personalized treatment strategies.

Citation

Kandemir H, Solakoglu Kahraman D, Aktas S, Unal OU, Cagiral S. Spatially Resolved FoxP3 Positive Lymphocyte Infiltration Within the Tumor Microenvironment Predicts Survival in Resected Pancreatic Ductal Adenocarcinoma. Discover Oncology. 2026. DOI: 10.1007/s12672-026-05195-7

 

Please sign in or register for FREE

If you are a registered user on Research Communities by Springer Nature, please sign in

Follow the Topic

Pancreatic Cancer
Life Sciences > Health Sciences > Clinical Medicine > Diseases > Gastrointestinal Diseases > Pancreatic disease > Pancreatic Cancer
Cancer Microenvironment
Life Sciences > Biological Sciences > Cancer Biology > Cancer Microenvironment
Tumour Immunology
Life Sciences > Biological Sciences > Immunology > Tumour Immunology
Regulatory T cells
Life Sciences > Biological Sciences > Anatomy > Haemic and Immune Systems > Immune system > Leukocytes > T cells > CD4-positive T cells > Regulatory T cells
Tumour Biomarkers
Life Sciences > Biological Sciences > Cancer Biology > Tumour Biomarkers
Disease-free Survival
Life Sciences > Health Sciences > Clinical Medicine > Prognosis > Disease-free Survival
  • Discover Oncology Discover Oncology

    This is a fully open access general oncology journal that aims to provide a unified forum for researchers and clinicians. The journal spans from basic and translational science, to preclinical, clinical, and epidemiology, and welcomes content that interfaces at all levels of cancer research.

Related Collections

With Collections, you can get published faster and increase your visibility.

Prostate Cancer - Advances in Diagnosis and Treatment

Prostate cancer remains one of the most prevalent malignancies among men globally, accounting for a significant proportion of cancer-related morbidity and mortality. With increasing life expectancy and improved diagnostic techniques, the incidence and detection rates of prostate cancer continue to rise, highlighting the critical need for advances in both diagnostic and therapeutic approaches. Recent breakthroughs in molecular biology, imaging technologies, and genetic profiling have significantly enhanced early detection and prognostic accuracy, allowing for more personalized patient care.

This Collection aims to present comprehensive insights into recent developments and cutting-edge research in prostate cancer diagnosis and treatment. Key topics include novel biomarkers for early detection, advances in imaging modalities such as multiparametric MRI and PET-PSMA, genomic and epigenetic factors influencing disease progression, and emerging therapeutic strategies including robotic-assisted surgery, radiotherapy advancements, immunotherapy, and precision medicine approaches. Additionally, the collection will explore mechanisms of resistance to therapy, the role of tumor microenvironment in disease evolution, and strategies to overcome treatment barriers. We invite contributions focusing on translational and clinical research, innovative diagnostic methods, therapeutic innovations, and studies that bridge fundamental discoveries with clinical application to improve patient outcomes in prostate cancer.

Keywords: prostate cancer, biomarkers, imaging, genomics, precision medicine, robotic surgery, radiotherapy, immunotherapy, tumor microenvironment

Publishing Model: Open Access

Deadline: Jan 04, 2027

The Evolving Landscape of Lung Cancer: Advances in Diagnosis, Treatment Strategies, and Simultaneous Care

Lung cancer remains one of the leading causes of cancer-related mortality worldwide, although recent years have seen remarkable progress in its clinical management. This Collection aims to deepen the evolving landscape of lung cancer through a comprehensive lens, including early diagnosis, molecular profiling (through liquid and tissue), therapeutic innovation, and simultaneous care strategies. The Collection will gather original research, reviews, short reports or letters and case studies focused on:

advances in early detection and biomarker discovery and development;

the evolution of precision oncology and molecular profiling;

updates on systemic therapies, including immunotherapy and targeted agents;

novel treatment combination strategies,

the increasing integration of simultaneous care into multidisciplinary models for improving patient outcomes.

Keywords: lung cancer; precision oncology; immunotherapy; molecular diagnostics; simultaneous care

Publishing Model: Open Access

Deadline: Dec 04, 2026